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Pharmacology Of Anti Ulcer Drugs Pdf Free

 
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MessagePosté le: Sam 15 Oct - 18:51 (2016)    Sujet du message: Pharmacology Of Anti Ulcer Drugs Pdf Free Répondre en citant




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Reduction of gastric acid secretion: H2 antihistaminics Cimetidine, Ranitidine, Famotidine, Roxatidine, Loxatidine etc Proton Pump Inhibitors Omeprazole, Lansoprazole, Pantoprazole, Rabeprazole etc Anti-cholinergics Pirenzepine, Propantheline etc Prostaglandin analogues Misoprostil, Enprostil, Rioprostil etc 2. (See "Overview and comparison of the proton pump inhibitors for the treatment of acid-related disorders", section on 'Treatment of acid-related disorders' and "Management and prognosis of the Zollinger-Ellison syndrome (gastrinoma)" and "Treatment regimens for Helicobacter pylori", section on 'Initial therapy' and "Treatment regimens for Helicobacter pylori".)Unlike H2RAs, tolerance does not occur for PPIs, but variable PPI metabolism may account for some difference in efficacy. Reduction of indomethacin-induced gastrointestinal blood loss by sodium salicylate in man. Similar to antacids, significant aluminum retention only occurs in patients with renal failure [3,35,36]. Hepatic effects of drugs used in the treatment of peptic ulcer disease. Ann Intern Med 1982; 97:242.Shields HM. (See "Overview and comparison of the proton pump inhibitors for the treatment of acid-related disorders".)Adverse effectsAdverse effects of PPIs are discussed in detail, separately. Always seek the advice of your own physician or other qualified health care professional regarding any medical questions or conditions. However, the salicylate from BSS will raise serum salicylate levels and can cause salicylate toxicity, and combination with other salicylate products should therefore be avoided.MISOPROSTOLMechanism of actionMisoprostol is a 15-deoxy-15-hydroxy-16-methyl analogue of prostaglandin E1.

Central nervous system (CNS) effects – H2RAs have been associated with CNS side-effects including confusion, restlessness, somnolence, agitation, headaches, dizziness, and, with prolonged therapy, hallucinations, focal twitching, and seizures [13,14,16]. Scand J Gastroenterol Suppl 1989; 164:224.Graham DY, Agrawal NM, Roth SH. (See "Aluminum toxicity in chronic kidney disease", section on 'Other medications'.)SUCRALFATEMechanism of actionSucralfate is a sulfated polysaccharide, sucrose octasulfate, complexed with aluminum hydroxide. (See "NSAIDs (including aspirin): Primary prevention of gastroduodenal toxicity", section on 'Misoprostol'.)POTASSIUM-COMPETITIVE ACID INHIBITORSPotassium-competitive acid inhibitors (PCABs) work by competing for potassium on the luminal side of the parietal cell, and cause rapid and reversible inhibition of pumps and therefore acid secretion [55]. Arch Intern Med 2000; 160:1455.Tarnawski A, Tanoue K, Santos AM, Sarfeh IJ. Use of UpToDate is subject to the Subscription and License Agreement. Gastroenterology 1991; 101:889.Sweeney JD, Hoernig LA. Is the quality of mucosal scar affected by treatment? Scand J Gastroenterol Suppl 1995; 210:9.Konturek SJ. Drugs 1988; 36:132.Nwokolo CU, Gavey CJ, Smith JT, Pounder RE.

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